The 9 Best Oils for Fine Hair Types

Having fine hair is no excuse anymore for having dry hair. With advancements in science and technology, there are many different oils out there to help your hair become more beautiful and saturated. We have the nine best oils for fine hair types, and you’re going to want to read this if it applies to you:

This luxury oil is formulated with sandalwood, cedarwood and argan oil and protects the hair from UV damage. It also happens to be one of our deputy beauty director’s all-time favorites. Here’s why.

“My hair is so fine that even some dry shampoos weigh it down (usually the wetter formulas that contain a lot of alcohol). But because I highlight it, and I don?t cut it very often, my ends are very dry. This beautiful-smelling stuff can go on damp or dry hair, but I like to use it once my hair is dry to smooth down frizz and add shine. Once I?ve blow-dried my hair, I squeeze out the tiniest little drop and rub it between my palms. Then I run it over the ends of my hair and whatever?s left I usually skim over my face-framing layers, which are fuzzy thanks to the fact I have a bad habit of pulling my hair back when it?s wet (which is a big no-no according to hairstylists). I also like to use it almost like a gel if I?m wearing my hair in a slicked-back ponytail or tight, low chignon.”

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Source: https://www.allure.com/gallery/best-oils-for-fine-hair

Dermatology scale validates quality of life

The quality of life can supposedly be determined by a dermatology scale. There are various scales out there that can study hyperpigmentation disorders among adults, and then show the person’s quality of life from the results. Who knew so much can be told about someone just from a possible skin disorder

Can having a skin condition impact the quality of your life? Absolutely, claim Boston University School of Medicine researchers who have set out to find the best tool to measure the impact on patients.

Several dermatology and disease-specific tools have been developed to measure the impact of skin disease including the widely used Dermatology Life Quality Index (DLQI) and the non-validated Skin Discoloration Impact Evaluation Questionnaire (SDIEQ). But the question remains: is one scale superior to the other, and/or easier to use?

In this study, BUSM researchers compared the DLQI with a short questionnaire (SDIEQ) to determine the impact of dark spots on a patients’ quality of life

After analysis of 321 adults with hyperpigmentation disorders using both scales the researchers found that DLQI and SDIEQ were similarly effective in measuring quality of life, however SDIEQ was simpler to use and less time consuming.

“Knowing how a condition impacts a patients’ quality of life is essential and a helpful guide in making treatment choices,” explained corresponding author Neelam Vashi, MD, assistant professor of dermatology at BUSM and director of the Boston University Cosmetic and Laser Center at Boston Medical Center. “Measuring health related quality of life is also important in patients when it comes to allocation of resources.”

Vashi added that further studies are needed to validate the use of this tool in different patient populations and potentially other disorders of pigmentation, such as vitiligo.

These findings appear as a Research Note in the Journal of Dermatology.

Source: https://www.sciencedaily.com/releases/2018/03/180320123448.htm?

Food allergy is triggered by perfect storm of genetics and skin exposure to infant wipes, dust and food

A lot of people you know may have a food allergy of some sort, as it is more common than not having any allergies. Some recent research has shown possible links between environmental factors and genetic factors coexisting to trigger this allergy. When it comes to children, this perfect storm is rather easy to concoct on it’s own:

Infant and childhood food allergy, whose cause has long been a mystery, has now been linked to a mix of environmental and genetic factors that must coexist to trigger the allergy, reports a new Northwestern Medicine study.

The factors contributing to food allergy include the genetics that alter skin absorbency, use of infant cleansing wipes that leave soap on the skin, skin exposure to allergens in dust and skin exposure to food from those providing infant care. Food allergy is triggered when these factors occur together.

“This is a recipe for developing food allergy,” said lead study author Joan Cook-Mills, a professor of allergy-immunology at Northwestern University Feinberg School of Medicine. “It’s a major advance in our understanding of how food allergy starts early in life.”

The paper will be published in the Journal of Allergy and Clinical Immunology on April 6.

Food allergies are on the rise and affect an estimated 4 to 6 percent of children in the U.S., according to the Centers for Disease Control and Prevention. The prevalence of reported food allergy increased 18 percent among children under age 18 years from 1997 to 2007. Recent data also show hospitalizations with diagnoses related to food allergies have increased among children.

Food allergy risk factors can be modified at home

The good news is factors leading to food allergy can be modified in the home environment, Cook-Mills said.

“Reduce baby’s skin exposure to the food allergens by washing your hands before handling the baby,” Cook-Mills said. “Limit use of infant wipes that leave soap on the skin. Rinse soap off with water like we used to do years ago. “

Scientist discovers the ‘perfect storm’ triggering food allergy

Cook-Mills made the discovery by using clinical evidence about food allergy in humans, the effects of food allergen and environmental allergen exposures and neonatal mice with genetic mutations that occur in humans.

Clinical evidence shows up to 35 percent of children with food allergies have atopic dermatitis and much of that is explained by at least three different gene mutations that reduce the skin barrier.

Cook-Mills used a neonatal mouse model with skin barrier mutations and tried exposing its skin to food allergens like peanuts. The peanuts alone had no effect.

“Then I thought about what are babies exposed to,” Cook-Mills recalled. “They are exposed to environmental allergens in dust in a home. They may not be eating food allergens as a newborn, but they are getting them on their skin. Say a sibling with peanut butter on her face kisses the baby. Or a parent is preparing food with peanuts and then handles the baby. “

Next, she read about skin research studies that delivered compounds through the skin by using soap. “I thought oh my gosh! That’s infant wipes!” Cook-Mills said.

The top skin layer is made of lipids (fats), and the soap in the wipes disrupts that barrier, Cook-Mills explained.

Skin problems that occur with skin barrier mutations may not be visible until long after a food allergy has already started. The neonatal mice with the mutations had normal-appearing skin, and the dry itchy skin of dermatitis did not develop until the mice were a few months old, the equivalent of a young adult in human years.

After the neonatal mice received three to four skin exposures of food and dust allergens for 40 minutes during a two-week period, they were given egg or peanut by mouth. The mice had allergic reactions at the site of skin exposure, allergic reactions in the intestine, and the severe allergic food reaction of anaphylaxis that is measured by decreased body temperature.

A skin barrier dysfunction was necessary for food allergy to develop in the mice, but there is a wide continuum of severe to mild skin dysfunction with eczema or atopic dermatitis, which in its mildest form may simply appear to be dry skin.

In patients with skin-barrier defects, there are changes in the proteins in the skin that are a result of mutations in the genes. These gene mutations in patients are primarily heterozygous, which means there is a mutation in one of the two copies of a gene.

Accordingly, in the preclinical studies, neonatal mice were also heterozygous for skin barrier mutations. The mice were co-exposed to food allergens such as egg and peanut proteins, allergens in dust (house dust mite or Alternaria alternata mold) and sodium lauryl sulfate, a soap present in infant cleansing wipes.

These novel animal studies provide a basis to test interventions that will more effectively block the development of food allergy in infants and children, Cook-Mills said.

She is currently studying molecular responses in the skin that are unique to this combination of genetics and skin exposures. The goal is to determine unique signals in the skin that occur during development of food allergy. This will lead to approaches to intervene with those skin signals and block the development of food allergy.

The study was supported by National Institutes of Health grants R01 HL124120, U01 AI131337 and R01 AI095282.

Source: https://www.sciencedaily.com/releases/2018/04/180406085504.htm?